AMS PART 1

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    Diabetes

    Diabetes Mellitus

    Metabolic disease

    Characterized by inadequate,absent insulin production

    Diabetes Mellitus

    Key Term

    The condition brought about by

    decreased insulin product ion, or the

    inability of the body cells to use

    insulin properly (which prevents the

    bodys cells from taking the simple

    sugar called glucose from the

    bloodstream)

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    Glucose

    Required as fuel for cellular metabolism

    Brains need for glucose parallels itsdemand for oxygen

    Insulin

    Hormone

    Produced by Islets of Langerhans inpancreas

    Required for sugar to enter most cells

    Brain does not require insulin to usesugar

    Insulin allows sugar to pass from the

    bloodstream into the cells.

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    Pancreas

    Located in

    retroperitoneal space

    Produces, releases

    Digestive enzymesinto duodenum

    Insulin, glucagon intoblood

    Islets of Langerhans

    Alpha cells

    Glucagon

    Raises blood sugar

    Beta cells

    Insulin

    Lowers blood sugar

    Type I Diabetes

    No insulin production

    Takes insulin injections

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    Diabetes is treated with injections of

    insulin or oral medications.

    Type II Diabetes

    Inadequate insulin production

    Increased tissue resistance to insulin effects

    Controlled with

    Diet

    Oral medications:

    Diabeta, Diabinese, Dymelor, Glucotrol,Micronase, Orinase, Tolinase, Glucophage

    Insulin injections as disease progresses

    Problems in Diabetes

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    Blood Sugar Imbalance Hyperglycemia

    Diabetic ketoacidosis (DKA) Hyperosmolar coma

    Hypoglycemia

    Hyperglycemia

    Key Term

    Hyperglycemia (high blood sugar) is

    a slow-onset condition from

    decreased insulin levels in people

    with diabetes.

    Hyperglycemia

    Causes

    Failure to take insulin

    Overeating, eating wrong diet

    Stress (fever, infection, emotional stress)

    New-onset diabetics usually presentwith an episode of hyperglycemia

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    Diabetic Ketoacidosis

    Usually Type I diabetic (no insulin)

    Blood sugar rises

    Kidneys try to remove excess sugar

    Urine production increases (polyuria)

    Patient becomes volume depleted

    Thirst (polydypsia)

    Tachycardia

    Hypotension

    Dry skin, mucous membranes

    Diabetic Ketoacidosis

    Cells cannot burn sugar; patient experienceshunger (polyphagia)

    Cells burn fat as alternative fuel

    Acidic ketone bodies produced

    Patient tries to correct acidosis; exhales CO2 Rapid, deep breathing (Kussmaul respirations)

    Exhaled ketone bodies produce nail-polishremover or fruity breath odor

    Diabetic Ketoacidosis

    Volume depletion

    Ketone body production (ketoacidosis)

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    Signs & Symptoms of

    Hyperglycemia

    Slow onset

    Nausea/vomiting

    Acetone odor on breath

    Increased urination/hunger/

    thirst

    Hyperosmolar Coma

    Usually Type II diabetic (inadequate insulin)

    Blood sugar rises

    Kidneys try to remove excess sugar

    Urine production increases (polyuria)

    Patient becomes volume depleted

    Thirst (polydypsia)

    Tachycardia

    Hypotension

    Dry skin, mucous membranes

    Hyperosmolar Coma

    Cells continue to burn sugar

    Acidic ketone bodies notproduced

    Nail-polish remover or fruity breath odor not

    present

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    Hyperosmolar Coma

    Severe volume depletion

    NO ketone body production

    Hyperglycemia

    Management

    Support ABCs

    Treat for hypovolemic shock

    Transport

    When in doubt, give sugar!

    Hypoglycemia

    Key Term

    Hypoglycemia (low blood sugar) is alife-threatening emergency for

    people with diabetes.

    It is the most common emergency

    for the diabetic patient.

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    Hypoglycemia

    Causes

    Insulin overdose Normal insulin use without eating

    Over-exercise

    Causes of Hypoglycemia

    Af ter taking too much insulin

    Vomiting

    Af ter unusual amount o f exercise

    Reduced sugar intake by not

    eating

    Hypoglycemia

    Alterations in consciousness;Seizures; Headache;Unusual Behavior

    Brain lacks adequate glucose

    Pale; Cool skin;Sweating; Tachycardia;Increased BP; Nausea

    Adrenal Glands release Epinephrine

    Blood Sugar Falls

    Pale, cool skin; sweating; nausea; tachycardia

    Is that why hypoglycemia sometimes is calledInsulin Shock?

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    Hypoglycemia

    Insulin shock isnt really shock

    Patient just looks shocky because ofepinephrine adrenals are releasing

    Hypoglycemia

    Can occur in non-diabetics

    Most common cause :EtOH on an empty stomach

    A patient is never, just drunk

    Signs & Symptoms of

    Hypoglycemia

    Rapid onset

    Intoxicated appearance, staggering,slurred speech, unconsciousness

    Cold, clammy skin

    Rapid heart rate

    Seizures (severe cases)

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    Unusual or bizarre behavior

    Anxiety

    Refusal to cooperate or

    combativeness

    Signs & Symptoms of

    Hypoglycemia

    Assess ing Diabetic

    Emergencies

    Perform initial assessment.

    Perform focused history and

    physical exam.

    Get SAMPLE history.

    Note any medical alert tags.

    Take baseline vital signs.

    Hypoglycemia Management

    Conscious patient

    Give sugar orally

    Unconscious patient

    Support ABCs

    Get ALS back-up for IV glucose

    When in doubt, Give Sugar!

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    When & how did it start?

    How long did it last?

    Complaints of other symptoms?

    Any trauma invol ved?

    Continued

    Assessing & Treating

    Diabetic Emergencies

    Perform focused history and

    physical exam.

    Any medical alert tags ?

    Has the patient seized?

    Fever?

    Interruptions in episode?

    Continued

    Assess ing & Treat ing

    Diabetic Emergencies

    Perform focused history and

    physical exam.

    Assessing & Treating

    Diabetic Emergencies

    Perform initial assessment.

    Maintain airway.

    Administer

    oxygen.

    Continued

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    Get a SAMPLE history.

    If the patient has a history of

    diabetes:

    When did patient last eat?

    Any medications ? Las t taken?

    Any other il lnesses?

    Can the patient swallow?

    Continued

    Assess ing & Treating

    Diabetic Emergencies

    Take baseline vital signs.

    In some areas, protocols direct the

    EMTB to tr eat the patient before

    getting vital signs.

    FOLLOW YOUR

    LOCAL PROTOCOL!

    Continued

    Assessing & Treating

    Diabetic Emergencies

    History of diabetes

    Altered ment al status

    Patient can swallow

    Continued

    Assess ing & Treating

    Diabetic Emergencies

    Give oral glucose if all of these

    conditions are met:

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    Reassess patient.

    If patient becomes unconscious,

    stop glucose administration

    immediately and secure the airway!

    If no improvement, consult medical

    direction.

    Continued

    Assess ing & Treating

    Diabetic Emergencies

    If patient is not awake enough to

    swallow:

    Secure airway.

    Adminis ter oxygen.

    Position appropriately.

    Request ALS & transpor t.

    Continued

    Assessing & Treating

    Diabetic Emergencies

    Obtain a blood glucose reading, if

    allowed by local protocols. Continued

    Assessing & Treating

    Diabetic Emergencies

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    Blood Glucose Readings

    80-120 mg/dl Normal

    60-80 mg/dl Moderate hypoglycemia

    Below 50 mg/d l Severe hypog lycemia

    Above 140 mg/dl Hyperglycemia

    Question results that are inconsistent

    with patients condition.

    Causes of Inaccurate Reading

    Meter not calibrated

    Low batteries in meter

    Improperly stored or expired

    test strip

    Insufficient blood on test strip

    Ask All Diabetics

    Have you eaten today?

    Have you taken your medication today?

    When in doubt, give Sugar!

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    Administration ofOral Glucose

    Squeeze glucose onto tongue depressor

    and place between cheek and gums.

    If the patient is alert enough, let her

    squeeze oral glucose into her mouth .

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    When the glucose is gone, remove

    tongue depressor and reassess patient.

    If the patient loses consciousness, remove

    tongue depressor, secure airway, and

    transport promptly.

    Oral Glucose

    Altered mental status

    with histor y of diabetes

    Indications

    Contraindications

    Unconsciousness

    Diabetic who has not

    taken insulin for days

    Inability to swallow

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    Oral Glucose

    Dosage One tube

    Oral Glucose

    Administrat ion

    Assure altered mental

    status with history of

    diabetes.

    Assure pat ient is

    conscious.

    Oral Glucose

    Administrat ion

    Admin is ter glucose on

    tongue depressorbetween cheek and gum

    or let patient self-

    administer.

    Perform ongoing

    assessment.

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    Oral Glucose

    None when given properly

    May be aspirated if given

    to patient without gag

    reflex

    Side Effects

    Actions Increases blood sugar

    Oral Glucose

    Reassessment Strategies

    If patient seizes or loses

    consciousness, remove

    tongue depressor and

    secure airway.

    Other Diabetes Complications

    Atherosclerosis

    Myocardial infarction

    CVA Peripheral vascular disease

    Blindness

    Renal failure

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    Other Diabetes Complications

    Diabetic Neuropathy

    Gangrene Increased silent myocardial infarction risk

    Silent MI

    Acute MI in diabetic can present withoutchest pain

    May resemble flu

    Manage sick diabetics as if critically illuntil proven otherwise