AMS PART 1
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Transcript of AMS PART 1
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Temple College EMS Professions
Diabetes
Diabetes Mellitus
Metabolic disease
Characterized by inadequate,absent insulin production
Diabetes Mellitus
Key Term
The condition brought about by
decreased insulin product ion, or the
inability of the body cells to use
insulin properly (which prevents the
bodys cells from taking the simple
sugar called glucose from the
bloodstream)
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Glucose
Required as fuel for cellular metabolism
Brains need for glucose parallels itsdemand for oxygen
Insulin
Hormone
Produced by Islets of Langerhans inpancreas
Required for sugar to enter most cells
Brain does not require insulin to usesugar
Insulin allows sugar to pass from the
bloodstream into the cells.
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Pancreas
Located in
retroperitoneal space
Produces, releases
Digestive enzymesinto duodenum
Insulin, glucagon intoblood
Islets of Langerhans
Alpha cells
Glucagon
Raises blood sugar
Beta cells
Insulin
Lowers blood sugar
Type I Diabetes
No insulin production
Takes insulin injections
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Diabetes is treated with injections of
insulin or oral medications.
Type II Diabetes
Inadequate insulin production
Increased tissue resistance to insulin effects
Controlled with
Diet
Oral medications:
Diabeta, Diabinese, Dymelor, Glucotrol,Micronase, Orinase, Tolinase, Glucophage
Insulin injections as disease progresses
Problems in Diabetes
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Blood Sugar Imbalance Hyperglycemia
Diabetic ketoacidosis (DKA) Hyperosmolar coma
Hypoglycemia
Hyperglycemia
Key Term
Hyperglycemia (high blood sugar) is
a slow-onset condition from
decreased insulin levels in people
with diabetes.
Hyperglycemia
Causes
Failure to take insulin
Overeating, eating wrong diet
Stress (fever, infection, emotional stress)
New-onset diabetics usually presentwith an episode of hyperglycemia
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Diabetic Ketoacidosis
Usually Type I diabetic (no insulin)
Blood sugar rises
Kidneys try to remove excess sugar
Urine production increases (polyuria)
Patient becomes volume depleted
Thirst (polydypsia)
Tachycardia
Hypotension
Dry skin, mucous membranes
Diabetic Ketoacidosis
Cells cannot burn sugar; patient experienceshunger (polyphagia)
Cells burn fat as alternative fuel
Acidic ketone bodies produced
Patient tries to correct acidosis; exhales CO2 Rapid, deep breathing (Kussmaul respirations)
Exhaled ketone bodies produce nail-polishremover or fruity breath odor
Diabetic Ketoacidosis
Volume depletion
Ketone body production (ketoacidosis)
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Signs & Symptoms of
Hyperglycemia
Slow onset
Nausea/vomiting
Acetone odor on breath
Increased urination/hunger/
thirst
Hyperosmolar Coma
Usually Type II diabetic (inadequate insulin)
Blood sugar rises
Kidneys try to remove excess sugar
Urine production increases (polyuria)
Patient becomes volume depleted
Thirst (polydypsia)
Tachycardia
Hypotension
Dry skin, mucous membranes
Hyperosmolar Coma
Cells continue to burn sugar
Acidic ketone bodies notproduced
Nail-polish remover or fruity breath odor not
present
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Hyperosmolar Coma
Severe volume depletion
NO ketone body production
Hyperglycemia
Management
Support ABCs
Treat for hypovolemic shock
Transport
When in doubt, give sugar!
Hypoglycemia
Key Term
Hypoglycemia (low blood sugar) is alife-threatening emergency for
people with diabetes.
It is the most common emergency
for the diabetic patient.
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Hypoglycemia
Causes
Insulin overdose Normal insulin use without eating
Over-exercise
Causes of Hypoglycemia
Af ter taking too much insulin
Vomiting
Af ter unusual amount o f exercise
Reduced sugar intake by not
eating
Hypoglycemia
Alterations in consciousness;Seizures; Headache;Unusual Behavior
Brain lacks adequate glucose
Pale; Cool skin;Sweating; Tachycardia;Increased BP; Nausea
Adrenal Glands release Epinephrine
Blood Sugar Falls
Pale, cool skin; sweating; nausea; tachycardia
Is that why hypoglycemia sometimes is calledInsulin Shock?
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Hypoglycemia
Insulin shock isnt really shock
Patient just looks shocky because ofepinephrine adrenals are releasing
Hypoglycemia
Can occur in non-diabetics
Most common cause :EtOH on an empty stomach
A patient is never, just drunk
Signs & Symptoms of
Hypoglycemia
Rapid onset
Intoxicated appearance, staggering,slurred speech, unconsciousness
Cold, clammy skin
Rapid heart rate
Seizures (severe cases)
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Unusual or bizarre behavior
Anxiety
Refusal to cooperate or
combativeness
Signs & Symptoms of
Hypoglycemia
Assess ing Diabetic
Emergencies
Perform initial assessment.
Perform focused history and
physical exam.
Get SAMPLE history.
Note any medical alert tags.
Take baseline vital signs.
Hypoglycemia Management
Conscious patient
Give sugar orally
Unconscious patient
Support ABCs
Get ALS back-up for IV glucose
When in doubt, Give Sugar!
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When & how did it start?
How long did it last?
Complaints of other symptoms?
Any trauma invol ved?
Continued
Assessing & Treating
Diabetic Emergencies
Perform focused history and
physical exam.
Any medical alert tags ?
Has the patient seized?
Fever?
Interruptions in episode?
Continued
Assess ing & Treat ing
Diabetic Emergencies
Perform focused history and
physical exam.
Assessing & Treating
Diabetic Emergencies
Perform initial assessment.
Maintain airway.
Administer
oxygen.
Continued
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Get a SAMPLE history.
If the patient has a history of
diabetes:
When did patient last eat?
Any medications ? Las t taken?
Any other il lnesses?
Can the patient swallow?
Continued
Assess ing & Treating
Diabetic Emergencies
Take baseline vital signs.
In some areas, protocols direct the
EMTB to tr eat the patient before
getting vital signs.
FOLLOW YOUR
LOCAL PROTOCOL!
Continued
Assessing & Treating
Diabetic Emergencies
History of diabetes
Altered ment al status
Patient can swallow
Continued
Assess ing & Treating
Diabetic Emergencies
Give oral glucose if all of these
conditions are met:
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Reassess patient.
If patient becomes unconscious,
stop glucose administration
immediately and secure the airway!
If no improvement, consult medical
direction.
Continued
Assess ing & Treating
Diabetic Emergencies
If patient is not awake enough to
swallow:
Secure airway.
Adminis ter oxygen.
Position appropriately.
Request ALS & transpor t.
Continued
Assessing & Treating
Diabetic Emergencies
Obtain a blood glucose reading, if
allowed by local protocols. Continued
Assessing & Treating
Diabetic Emergencies
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Blood Glucose Readings
80-120 mg/dl Normal
60-80 mg/dl Moderate hypoglycemia
Below 50 mg/d l Severe hypog lycemia
Above 140 mg/dl Hyperglycemia
Question results that are inconsistent
with patients condition.
Causes of Inaccurate Reading
Meter not calibrated
Low batteries in meter
Improperly stored or expired
test strip
Insufficient blood on test strip
Ask All Diabetics
Have you eaten today?
Have you taken your medication today?
When in doubt, give Sugar!
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Administration ofOral Glucose
Squeeze glucose onto tongue depressor
and place between cheek and gums.
If the patient is alert enough, let her
squeeze oral glucose into her mouth .
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When the glucose is gone, remove
tongue depressor and reassess patient.
If the patient loses consciousness, remove
tongue depressor, secure airway, and
transport promptly.
Oral Glucose
Altered mental status
with histor y of diabetes
Indications
Contraindications
Unconsciousness
Diabetic who has not
taken insulin for days
Inability to swallow
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Oral Glucose
Dosage One tube
Oral Glucose
Administrat ion
Assure altered mental
status with history of
diabetes.
Assure pat ient is
conscious.
Oral Glucose
Administrat ion
Admin is ter glucose on
tongue depressorbetween cheek and gum
or let patient self-
administer.
Perform ongoing
assessment.
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Oral Glucose
None when given properly
May be aspirated if given
to patient without gag
reflex
Side Effects
Actions Increases blood sugar
Oral Glucose
Reassessment Strategies
If patient seizes or loses
consciousness, remove
tongue depressor and
secure airway.
Other Diabetes Complications
Atherosclerosis
Myocardial infarction
CVA Peripheral vascular disease
Blindness
Renal failure
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Other Diabetes Complications
Diabetic Neuropathy
Gangrene Increased silent myocardial infarction risk
Silent MI
Acute MI in diabetic can present withoutchest pain
May resemble flu
Manage sick diabetics as if critically illuntil proven otherwise